Clarifying the role of breastfeeding among women with a BRCA1 or BRCA2 mutation may be helpful for risk assessment and for recommendations regarding prevention. More than 12 % of women will be diagnosed with breast cancer in their lifetime. Purpose Young women who have been identified as carrying a deleterious mutation in BRCA1 or BRCA2 face a unique set of challenges related to managing cancer risk during a demographically-dense stage of life.
The recently identified genes BRCA1 and BRCA2 appear to account for the majority of hereditary breast cancer in US and European populations. BRCA1 and BRCA2 are human genes that produce tumor suppressor proteins.These proteins help repair damaged DNA and, therefore, play a role in ensuring the stability of each cell’s genetic material. Original Article from The New England Journal of Medicine — Maintenance Olaparib for Germline BRCA-Mutated Metastatic Pancreatic Cancer Breastfeeding has been inversely related to breast cancer risk in the general population. When either of these genes is mutated, or altered, such that its protein product is not made or does not function correctly, DNA damage may not be repaired properly. We present an updated analysis of breastfeeding and risk of breast cancer using a large matched sample of BRCA mutation carriers. BRCA1 was amongst the first cancer predisposition genes discovered, and it has been studied for over 20 years. Although there have been tremendous advances in elucidating genetic risk factors underlying both familial and sporadic breast cancer, much of the genetic contribution to breast cancer etiology remains unknown. Both of these genes have already been localized and isolate; however, the exact functions of their proteins are not clear yet. BRCA1 is a gene on chromosome 17 that normally helps to suppress cell growth.
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The recently identified genes BRCA1 and BRCA2 appear to account for the majority of hereditary breast cancer in US and European populations. BRCA1 and BRCA2 are human genes that produce tumor suppressor proteins.These proteins help repair damaged DNA and, therefore, play a role in ensuring the stability of each cell’s genetic material. Original Article from The New England Journal of Medicine — Maintenance Olaparib for Germline BRCA-Mutated Metastatic Pancreatic Cancer Breastfeeding has been inversely related to breast cancer risk in the general population. When either of these genes is mutated, or altered, such that its protein product is not made or does not function correctly, DNA damage may not be repaired properly. We present an updated analysis of breastfeeding and risk of breast cancer using a large matched sample of BRCA mutation carriers. BRCA1 was amongst the first cancer predisposition genes discovered, and it has been studied for over 20 years. Although there have been tremendous advances in elucidating genetic risk factors underlying both familial and sporadic breast cancer, much of the genetic contribution to breast cancer etiology remains unknown. Both of these genes have already been localized and isolate; however, the exact functions of their proteins are not clear yet. BRCA1 is a gene on chromosome 17 that normally helps to suppress cell growth.
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